Could You please explain to me why 6 weeks on amoxycillin, clarytromycine and metronidazolei is not enough to get rid of cpni?
It will attack all 3 forms of cpn and it's lifecycle is 50-70 hours so 6 weeks should be absolutely enough?
This is what my doctor says. In order to avoid future problems could You please help me what to say to my doctor to convince him for longer (1 year) therapy?
I have cpn iggi 105 (5 times more than limit).
Looking forward for Your help!
Lucas.
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Hi Lucas,it`s simple.a) You
Hi Lucas,
it`s simple.a) You don`t want to suffer from treatment-failures.
b) Your dotor needs a chance to learn, so "feed" him with all the knowledge you can find here.
If he doesn`t want to change his mind, find another doctor, there are good ones around.
I wish you all the best.
sphinx
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sphinx /On Wheldon CApi ,14 th May 2008, currently: Doxyi 200 ,ACC 1200 plus supplementsi ,RRMSi since 92, SPMSi 06, EDSS 7 ,Azi 3 times a week, Tinii 2 days ,1 tab only
I think I need medical
Here is a medical
Here is a medical explanation from my husband's website:
"What is the life-cycle of C pneumoniae?
Simplifying slightly, C pneumoniae has two major phases to its life cycle. The infectious form is called the Elementary Body (EB). This spore-like form transmits the organism from person to person and, within a person, from cell to cell. The EB is metabolically inert; it has an external membrane which includes potentially unstable proteins. When it attaches to a susceptible host cell a sudden change takes place; the proteins which are embedded in its external membrane become loose and the host cell wall closes behind the entering EB. Entry may be controlled by subversional proteins injected into the host cell via tiny syringes and needles (The type III secretory system. Proteins associated with the TTSS have been found in the EB proteome.)
Once inside the host cell, the organism expands to become the Reticular Body (RB). It efficiently uses host membrane to make its own environment within a cell. Then it takes up its position around the host's mitochondria to steal the energy-rich molecules fabricated by these organelles, probably by means of small tubes. The RB then begins to control the direction of the host-cell metabolism, again probably using microscopic syringes and needles. Its own nuclear material eventually divides into many separate individuals, which then condense into EBs. The host cell bursts, scattering EBs into the extracellular milieu. This is the picture in cell-culture and is the likely picture in acute infectionsi.
In chronic infections a different pathway is taken. Under pressure from host defences the organism enters into persistent state, where its metabolic processes are diminished. The organism in this state is called the Cryptic Body (CB). This chronic unresolved infection - which can last for several decades - can initiate the malign process of autoimmunityi. To a large extent the form of the disease depends on the host's genetic inheritance. This is why many of the chronic disease forms caused by infections with Chl pneumoniae tend to have inherited characteristics. An excellent and readable account of how peristent unresolved infections can initiate chronic diseasesi with autoimmune aspects can be found in this dissertation by Tiina Sävykoski: http://herkules.oulu.fi/isbn9514269853/html."
This is the whole site and is as relevant to all diseases which have CPni as a cause, not just MS:http://www.davidwheldon.co.uk/ms-treatment.html ............Sarah
An Itinerary in Light and Shadow
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Lucas - The information
Lucas - The information that Sarah gave you is excellent!
You might add to it by printing out the full patent by Dr. Charles Strattoni at Vanderbilt University and giving that also to your doctor. It's long, detailed and full of impressive science.
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Daisy - Husband on CAPi 5/07. "When Going Thru Hell, Just Keep Going", Winston Churchill
Lucas,In addition to
Lucas,
In addition to Sarah's excellent information... you may also want to review and print out this:
Clikc on the "Cpnii Handbook" tab, then to:
1. Chlamydia Pneumoniae in Human Disease, then, click
2. How Chlamydia Pneumoniae Causes such a Plethoria of Diseasesii.
It shows how the CPN bacteria is carried to our other organs and bone marrow of the body.
Jeanneroz
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JeanneRoz~CPNi diagnosed & started protocol 4/2007, also HHV6, EBVi. CFIDSi/FM diagnosed: 6/07; 100mg/doxyi/BID ~ 250 mg AZITH M/W/F ~
1st Tinii pulse 4/17/08- 1 250 mg. tab for 2 days. 3RD pulse 7/7/2008=500 mgTINI:4dayJeanne, There is something
Jeanne, There is something amiss with the link above. Did not work for me, page not found.
Louise
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Louise-CFSi, CPN+/Bb+ Wheldon CAPi 6/07, Cholestyramine1-2pksHSforPorphoria& Endotoxinsi, Doxy100daily,Roxi300BID,Tini500mgBIDpulses,VitD3-4000IU,MagnascentIodine,{S.O.D.3/QD[KAL Brand],+Pyruvate3.75G+SAM-eForEnergy}
Louise,You are right... Try
Louise,
You are right...
Try going to the "Cpni Handbook" tab, then to:
1. Chlamydia Pneumoniae in Human Disease, then, click
2. How Chlamydia Pneumoniae Causes such a Plethoria of Diseasesi.
I can't get the link to complete either, sorry!
Jeanneroz
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JeanneRoz~CPNi diagnosed & started protocol 4/2007, also HHV6, EBVi. CFIDSi/FM diagnosed: 6/07; 100mg/doxyi/BID ~ 250 mg AZITH M/W/F ~
1st Tinii pulse 4/17/08- 1 250 mg. tab for 2 days. 3RD pulse 7/7/2008=500 mgTINI:4dayThank You so much! This
Thank You so much! This article "How Chlamydia Pneumoniae Causes such a Plethoria of Diseasesi" is so true!!
I feel imflammed very often, I tested immunoglobulines and my iggi, igm, iga are at lower limit. I feel tired often, I have heart pressure changes and other sympthoms of cpni infection described in this article. I am sure I have chronic cpn infection from 12 years.
I will take herbs and supplement for 4 weeks to improve my overall codition and than I go to see my doctor again with printed papers You recomended to talk about starting CAPi.
It's a pity we have this damn bacteria and there is no chance to get rid of it in six weeks isn't it? :)
Thanks again for link to this article.