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Ability of bacteria to persist desptite treatment or immune function.

The Elephant(s) in My Room – Blog Update

I haven't posted much and my last blog update was in May of this year. I wish the reasons were because I was enjoying a recovery and had my health back, but it's not.   I now join the ranks of some of the others here who came to this site about the time I did.  


Last week I had Bioenergetic Testing done which indicated I have:  Bartonella Henselae, Babesia Nosodes, Mycoplasma Fermentes, Borrelia Burgdorferi and high levels of systemic yeast (as well as EBVi, HHV-6 and CPNi) -- "my elephants" and reasons for not recovering. 


solutition to stubburn bacterial infections on the news yesterday

cpni is not mentioned but tuberculosis it. It make alot of sense.<

Development of Chlamydia pneumoniae inclusion

Interesting, yet kinda creepy knowing this is what is going on inside us.

   Development of Chlamydia pneumoniae inclusion




3 Years, 8 Months, 2 weeks and 2 days.....(Revised 2/14/2011)

It has been 3 years, 8 months, 2 weeks and 2 days since I started CAPi<i<.   I have reached a point, not by choice, but rather by necessity, that I have had to back off the protocol for a while. 

Quick Update

Just a quick update on the progress so far on the new treatment plan.

My naturopath consulted with a doc who has had a lot of success treating Lyme and co-infectionsi with natural remedies. He recommended Byron White's formulas. I am currently ramping up on A-Bart for Bartonella.

According to this Lyme expert, these infections take turns being active in the body. He told my doctor to observe symptoms to determine which one is on top of the stack. He compares it to layers. That is the one to go after. It is all about timing.

I began A-Bart about a week ago and currently taking up to 10 drops. Supposed to get up to 20 drops. It smells like barbeque sauce--very strong garlic smell. I have had Tinnitus">i and night sweats-also waking up in the middle of the night .

Cpn Theory: Cpn Metabolism

Cpni Theory: Cpn Metabolism

Chlamydia Pneumonia is an obligate intracellulari pathogen that relies on the host cell for energy. It is able to obtain energy through multiple mechanisms. The primary mechanism is exchanging ATP for ADP with the host cell mitochondria. ATP and ADP are exchanged using a proton pump which may require a lower chlamydial pH than that of the host cell. Probably the use of protons to exchange ATP for ADP and the subsequent use of ATP lead to a homeostasis of Cpn pH so this should be able continue under normal circumstances. This pH level and reliance on ATP/ADP exchange probably represents the reticulate body phase in chlamydial development. At this pH level Cpn can probably make the proteins needed for replication.

Who out there is functional? (Non-Ms'ers)

For the benefit of perspective......I am curious.  We get varied responses on physical reactions to pulses, ABXi, etc.; BUT,  in the big picture, how many of you ( on a day-to-day basis), are still functioning below normal.... or non-functional?   How many are unable to work, drive, take care of the home, cook, etc.?

Some of you speak of your pulses taking you down, but yet have the physical capabilities of going to the gym, carrying out day-to-day activities and working.  (Which, by the way, is fantastic)  This query is in no way meant as an offense to anyone.  

 Since I am unable to do a poll, is anyone willing to share?


How long on CAP:



Physical levels of endurance: 



Cpn Theory: Mechanism of Resistance

Cpni Theory: Mechanism of Resistance

Chlamydia Pneumonia is an obligate intracellulari pathogen associated with a variety of chronic illnesses. Like many other pathogens, it has evolved mechanisms that allow it to survive in a hostile host environment and incidentally may be directly or indirectly responsible for many chronic illnesses.

Persister Cells and the Paradox of Chronic Infections

Hi, everybody,

 I hardly ever come here these days, so I apologise if this article has already been posted, but just in case it hasn't... here it is


Persister Cells and the Paradox of Chronic Infections

Dormant persister cells are tolerant to antibioticsi and are largely responsible for recalcitrance of chronic infections Kim Lewis Kim Lewis is a Professor in the Department of Biology, and Director of the Antimicrobial Discovery Center, Northeastern University, Boston.

Cpn theory: Effect of pH on Cpn

Cpni theory: Effect of pH on Cpn

Layers of problems

First, thanks for the encouragement from everyone. It's been a really bad week - I would say I was suicidal several times. As with many of you, so much is going on in addition to whatever the CPNi is doing to my body. I am taking appropriate meds for it all and seeing my docs.
While laying in bed with too much time to think, these are the layers of problems I came up with.
1. General Anxiey Disorder - diagnosed years ago while raising and homeschooling five children ages ten and under. Marriage problems added to the anxiety.
2. Panic attacks - I had some even as a child, but didn't know what they were.

Does it ever get Better?

I am seriously, severely depressed and I'm getting really depressed about it.   I've only been on CAPi for a few months and already I'm tired of it ready to give up. When do I start feeling better? Where do CFSi/Fibro  symptoms end and just plain old depression kick in? I'm getting some of the best care possible and on tons of meds and support. Yet I can't make it through the day without falling apart.

Biofilms - Clinical Implications of, by Dr. Stephen Olmstead, Chief Science Officer for Prothera and Klaire Labs -

On February 26, 2009, Dr. Stephen Olmstead, Chief Science Officer for ProThera® and Klaire Labs™, gave a lecture to the Physicians’ Roundtable meeting in Greenville, North Carolina on gastrointestinal biofilm.
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