Hi Lucas,

it`s simple.a) You don`t want to suffer from treatment-failures.

                  b) Your dotor needs a chance to learn, so "feed" him with all the knowledge you can find here.

If he doesn`t want to change his mind, find  another doctor, there are good ones around.

I wish you all  the best.

sphinx

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sphinx /On Wheldon CApi [4] ,14 th May 2008, currently/ Minoi [5] 200 ,ACC 1200  plus supplementsi [6] ,RRMSi [7] since 92, SPMSi [8] 06,  EDSS 7  ,Azi 3 times a week, 3  Tinii [9] pulses

Here is a medical explanation from my husband's website:

"What is the life-cycle of C pneumoniae?

Simplifying slightly, C pneumoniae has two major phases to its life cycle. The infectious form is called the Elementary Body (EB). This spore-like form transmits the organism from person to person and, within a person, from cell to cell. The EB is metabolically inert; it has an external membrane which includes potentially unstable proteins. When it attaches to a susceptible host cell a sudden change takes place; the proteins which are embedded in its external membrane become loose and the host cell wall closes behind the entering EB. Entry may be controlled by subversional proteins injected into the host cell via tiny syringes and needles (The type III secretory system. Proteins associated with the TTSS have been found in the EB proteome.)

Once inside the host cell, the organism expands to become the Reticular Body (RB). It efficiently uses host membrane to make its own environment within a cell. Then it takes up its position around the host's mitochondria to steal the energy-rich molecules fabricated by these organelles, probably by means of small tubes. The RB then begins to control the direction of the host-cell metabolism, again probably using microscopic syringes and needles. Its own nuclear material eventually divides into many separate individuals, which then condense into EBs. The host cell bursts, scattering EBs into the extracellular milieu. This is the picture in cell-culture and is the likely picture in acute infectionsi [10].

In chronic infections a different pathway is taken. Under pressure from host defences the organism enters into persistent state, where its metabolic processes are diminished. The organism in this state is called the Cryptic Body (CB). This chronic unresolved infection - which can last for several decades - can initiate the malign process of autoimmunityi [11]. To a large extent the form of the disease depends on the host's genetic inheritance. This is why many of the chronic disease forms caused by infections with Chl pneumoniae tend to have inherited characteristics. An excellent and readable account of how peristent unresolved infections can initiate chronic diseasesi [12] with autoimmune aspects can be found in this dissertation by Tiina Sävykoski: http://herkules.oulu.fi/isbn9514269853/html [13]."

This is the whole site and is as relevant to all diseases which have CPni [2] as a cause, not just MS:http://www.davidwheldon.co.uk/ms-treatment.html [14] ............Sarah

An Itinerary in Light and Shadow

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Lucas - The information that Sarah gave you is excellent! 

You might add to it by printing out the full patent by Dr. Charles Strattoni [17] at Vanderbilt University and giving that also to your doctor.  It's long, detailed and full of impressive science. 

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Daisy - Husband on CAPi [4] 5/07.  "When Going Thru Hell, Just Keep Going", Winston Churchill

Lucas,

In addition to Sarah's excellent information... you may also want to review and print out this:

Clikc on  the "Cpni [2]i [2] Handbook" tab, then to:

1. Chlamydia Pneumoniae in Human Disease, then, click

2.  How Chlamydia Pneumoniae Causes such a Plethoria of Diseasesi [12]i [12].

It shows how the CPN bacteria is carried to our other organs and bone marrow of the body. 

Jeanneroz

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JeanneRoz~CPNi [2] diagnosed & started protocol 4/2007, also HHV6, EBVi [18]. CFIDSi [19]/FM diagnosed: 6/07; 100mg/doxyi [20]/BID ~ 250 mg AZITH M/W/F ~1st Tinii [9] pulse 4/17/08- 1 250 mg. tab for 2 days. Pulse 5: 9/28/08, 250 mg TINI BID, 3 days. Sup

Jeanne, There is something amiss with the link above.  Did not work for me, page not found.

Louise

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Louise  CFSi [21], CPN+/Bb+,Wheldon CAPi [4] 6/07, Cholestyramine 1-2 pks @ HS for Porphyriai [22] & Endotoxinsi [23] PRN, Doxyi [20] 200daily, Roxi 300BID, Tini500BIDx14day pulses,VitD3-10,000IU, Iodoral 25mg, {S.O.D.3/QD[KAL Brand], Pyruvate 3.75G, SAM-e For Energy Support

Louise,

You are right...

Try going to the "Cpni [2] Handbook" tab, then to:

1. Chlamydia Pneumoniae in Human Disease, then, click

2.  How Chlamydia Pneumoniae Causes such a Plethoria of Diseasesi [12].

I can't get the link to complete either, sorry! 

Jeanneroz

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JeanneRoz~CPNi [2] diagnosed & started protocol 4/2007, also HHV6, EBVi [18]. CFIDSi [19]/FM diagnosed: 6/07; 100mg/doxyi [20]/BID ~ 250 mg AZITH M/W/F ~1st Tinii [9] pulse 4/17/08- 1 250 mg. tab for 2 days. Pulse 5: 9/28/08, 250 mg TINI BID, 3 days. Sup

Thank You so much! This article "How Chlamydia Pneumoniae Causes such a Plethoria of Diseasesi [12]" is so true!!

I feel imflammed very often, I tested immunoglobulines and my iggi [3], igm, iga are at lower limit. I feel tired often, I have heart pressure changes and other sympthoms of cpni [2] infection described in this article. I am sure I have chronic cpn infection from 12 years.

I will take herbs and supplement for 4 weeks to improve my overall codition and than I go to see my doctor  again with printed papers You recomended to talk about starting CAPi [4].

It's a pity we have this damn bacteria and there is no chance to get rid of it in six weeks isn't it? :)

Thanks again for link to this article.