Iron deprivation is normally associated with cpni [1]. A number of us have to supplement.

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The difference between what we do and what we are capable of doing would suffice to solve most of the world’s problems. Mohandas Gandhi

Macintosh, 

Sounds like I have opposite problem - I have too high level of iron. This is why I would like to know if this could be assosiated with cpni [1] as well.

Iron deprivation if I understand correct leads to too low level of iron in blood and necessity of supplementation?

Cpni [1] is an iron using bacterium. It uses it actively in it's replication. So in addition to the Cpn using up some of your free iron, the immunei [2] system starts sequestering free iron in cells to keep it from the bacterium, leaving less free circulating iron. Before I found out I was positive for Cpn I was being treated for low iron (unusual for men). As the Cpn infection improved, my iron normalized. I don't know if there might be some reaction that causes higher iron.

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CAPi [3] for Cpni [1] 11/04. Dx: 25yrs CFSi [4] & FMSi [5]. Currently: 150mg BID Roxithromycin, Doxycycline 100mg BID, Tinii [6] 1000mg/day pulses; Vit D2000 units, T4 & T3

Jim,

I have normal Total iron-binding capacity, but higher Transferrin saturation which might be a sign of iron deficiency.

On the other hand I have high iron.

I heard that iron level is higher in the morning (I made a test at 8am) and iron absorption is increased when supplementing vitamin C (I took 1,5 gram day before test). some sources say it is also increased after using fluorine and I was taking ofloxacine (fluoroquinolone)for 30 days.

Is it possible to have iron deficiency and high iron level in blood after ofloxacine and vit C?What do You think about it?

My  labs last month  indicated  my Ferritin level was at 42 (optimumum should be between 70-90 per this [7] article and Dr.)   I didn't realize CPNi [1] was an iron-using bacteria.   

I have been concentrating on getting my adrenals and thyroid levels optimized (which is tricky as others of you may know with CPN and thyroid problems) and read where low iron  attributes to fatigue and hypothyroidism per the above  link and Dr.

I guess now knowing CPN steals iron  makes even more sense as to the fatigue -- since the buggars are stealing it!  All these years of giving myself B12 shots were just helping them along!

I definitely will be supplementing my  iron -- I have visions of feeding time at a Koi pond, LOL.

Jeanneroz

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JeanneRoz~CPNi [1] diagnosed & started protocol 4/2007, also HHV6, EBVi [8]. CFIDSi [9]/FM diagnosed: 6/07; 100mg/doxyi [10]/BID ~ 250 mg AZITH M/W/F ~1st Tinii [6] pulse 4/17/08- 1 250 mg. tab for 2 days. Pulse 5: 9/28/08, 250 mg TINI BID, 3 days. Sup

Mairj, you have high iron and high transferrin which indicate iron overload, not deficiency. Normal TIBC is still compatible with hemochromatosis, although it is usually low. Alcohol consumption, taking iron with with vitamin C or iron consumed without anemia will overcome the intestinal barrier which prevents excessive iron absorption from food. Food rich in iron causes temporary elevation of serum iron, so blood should be taken fasting. Barbara

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Cured of multiple sclerosisi [11], stopped the Wheldon's protocol in Nov,2008. EDSSi [12] 0 for over 5 years.

I have hemochromatosis & even with the CPni [1], my iron storage is high.  Not so dangerously though that I have to have some blood drained out.  Now, when the critters are gone, who knows; but one thing at a time.

hope you are doing well with the balancing act, I know how that alone, can be exhausting!

peace

 

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CFIDSi [9]/ME 32 yrs, FMSi [5], IBSi [13], EBVi [8], CMV, Cpni [1], chronic insomnia, Lymes, HME, Natural HRT peri-M, NAC 3 gm, 6-07 Doxy 200 mg day pm, Azith 375 mg M/W/Fday, Pulse#15 750 mg X 5 days 11-1-08

I need to check if this is hemochromatosis. Also in hemolytic anemia serum iron and transferine saturation is high with tibc normal and higher bilirubine . Lets check what doctors think. I need to do some additional tests I suppose.

Thanks a lot for Your posts!