Thoughts on staph

I was reading about staph infection, osteomylitis and mssa vs mrsa and the reference i read showed that chronic staph antibioticsi [1] can include minocin combined with rifampin. I was wondering... does this mean that doxyi [2] and rifampin would have an effect against staph as well? maybe just not as potent?

How would dr's even know if a person had a staph infection vs staph colonization anyway? It seems DR's only pay attention to MRSA but if a person is penacillin allergic they would never develop MSRA from thier chronic staph infection thus it would get ignored and pawned off as colonization or normal flora no?

Furthermore... what if peoples staph infectionsi [3] were in places that couldnt be swabbed for example, such as bones or organs or jaw or teeth... how would a dr ever know if a person had a staph infection that was making them sick?

How do we even know that rifampin and a tetracycline are treating not only cpni [4] but staph as well ?

I look at the situation with montel williams who was clearly diagnosed with MS. I clearly remember him raising awareness on MRSA and I also clearly remember him mentioning the importance of oral dental health and cavitations and fillings etc. I just find it odd that if there is any conspiracy with montel and the drug companies it would be more likely that he was treated with superdrugs for resistant staph infection as opposed to cpn no? Why would he care about MRSA unless for some reason this infection has had some personal impact on him?

As well... I think its interesting that the article just posted in this section illustrates minocen being used as well.

Anyway just my thoughts... has anyone else considered staph as a key player in MS ?

CPNi [4] pcri [5]i [5] and antibody positive , treating MSi [6], ~~CFSi [7]i [7]~~, TMJ, trigeminal neuralgia, ~~IBSi [8]i [8]~~ neutropenia, pus found in facial bone, ~~Doxyi [2] 100x2~~,Doxy 200x2 zithro 250x1 alternate days. Metroi [9]i [9] pulses each month.

This would seem to [10]

Submitted by Sarah on Mon, 2008-05-26 13:36.

This would seem to indicate that a staphyococcal infection might actually act as an immunosuppressant for someone with multiple sclerosis. Maybe the MSi [6] becomes worse when a staph infection is got rid of, bearing in mind that treating a staph infection doesn't take years, but rather days:

http://www.jem.org/cgi/content/abstract/jem.20051681v1 [11]............Sarah

An Itinerary in Light and Shadow...........

Completed Stratton/Wheldon regime for aggressive secondary progressive MS in June 2007, after nearly four years, three of which intermittent. Still slowly improving and no exacerbation since starting. EDSSi [12] was 7, now 2, less on a good day.

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Completed Stratton/Wheldon regime for aggressive secondary progressive MSi [6] in June 2007, after nearly four years, three of which intermittent. Still slowly improving and no exacerbation since starting. EDSSi [12] was 7, now 2, less on a good day.

Oh, great. Well, at least I

Submitted by Minai on Tue, 2008-05-27 08:36.

Oh, great. Well, at least I might catch it again...husband's nose is broken out, again. Prompted me to ask and he said he's had lesions on his head that started developing when he was using the bactroban in his nose. Interesting to say the least. RRMSi [13], diagnosed 2/04. NACi [14] 4/06. Started Wheldon/Stratton regime 8/30/06. Doxycycline, 8/06, Azith, 10/06. Switched to Roxithromycin 11/06. Psuedo relapse/die-off with hospitalization 1/07. GAD-enhanced MRI of brain and spine shows NO NEW DISEASE ACTIVITY. LDNi [15] 4/07. 1st Tinidazole Pulse, 8/11/07. Keflex 2/08. IV Rocephini [16] 3/08. IV Clindamycin 5/08. USA

Clammed, My thought is that

Submitted by Louise on Tue, 2008-05-27 10:19.

Clammed, My thought is that it makes sense that when you take any antibacterial substance correctly that is appropriate for any particular organism in the proper dosage for the appropriate length of time then you certainly may impact organisms that you did not even know are there then as a result.

From my point of view, things are not as specific as you might think from my reading of your questions, here an on other threads.

From my perspective this is a good thing. How can we really know what we have that is contributing to our manifestations of illness? It speaks to the appropriatness of emperical treatment for a growing list of chronic degenerative and life inhibiting conditions, again from my personal opinion only.

Best Regards to you,

Louise

CFS. CPnPositive. BbPositive. WheldonCAP began6/24/07. NACi [14],Doxyi [2], Roxi,FullTiniPulses. Intermittent Cholestyramine,1-2packets, at bedtime,most often with pulses,and as needed, for Phorphoria & liposacaride Endotoxini [17] Die-OffExperiences.

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Louise CFSi [7], CPN+/Bb+,Wheldon CAPi [18] 6/07, Cholestyramine 1-2 pks @ HS for Porphyriai [19] & Endotoxinsi [20] PRN, Doxyi [2] 200daily, Roxi 300BID, Tini500BIDx14day pulses,VitD3-10,000IU, Iodoral 25mg, {S.O.D.3/QD[KAL Brand], Pyruvate 3.75G, SAM-e For Energy Support

Sarah, well maybe its

Submitted by clammed_up on Tue, 2008-05-27 09:54.

Sarah, well maybe its possible then that our body produces staph infection in response to cpni [4], in an attempt to prevent autoimmune encephalitisi [21]? Though my gut thinks there is some cofounding variable overlooked in that study, but I dont really know to be honest. Good info though thanks

I think staph would be fairly quick and easy to treat if it wernt in the bone yes, but staph in the bone if in a place that is necrotic and abxi [1] cant reach... how would a person ever get better? I think the drugs would clear the systemic infection from the body but how would the drugs rid the necrotic bone of the infection? Would a person just need abxi [22] forever?

Also, I know from personal experience that not all bone infection shows up on film. I had experimental surgery once that pulled pus out of my bone and it didnt even show up as an inflamatory response on film. Also.. it cultured as absolutly nothing being that I was on antibiotics at the time of surgery.

So, a person could technically have a bone infection without it showing up on film.

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CPNi [4] pcri [5] and antibody positive , treating MSi [6], ~~CFSi [7]~~, TMJ, trigeminal neuralgia, ~~IBSi [8]~~ neutropenia, pus found in facial bone, ~~Doxyi [2] 100x2~~,Doxy 200x2 zithro 250x1 alternate days. Metroi [9] pulses each month.